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Axonal Pathology In MS

  1. A Magnetization Transfer MRI study of deep Gray Matter involvement in Multiple Sclerosis
    J NeuroImaging 2006 Oct;16(4):302-10

  2. MR Spectroscopic evidence for Thalamic and Hippocampal, but not Cortical, damage in Multiple Sclerosis
    Magn Reson Med 2006 Mar;55(3):478-83

  3. MRI evidence for Multiple Sclerosis as a diffuse disease of the Central Nervous System
    J Neurol 2005 Oct;252 Suppl 5:v16-v24

  4. Emergence of Thalamic Magnetization Transfer Ratio abnormality in early Relapsing/Remitting Multiple Sclerosis
    Mult Scler 2005 Jun;11(3):276-81

  5. Axonal damage accumulates in the Progressive phase of Multiple Sclerosis: three year follow up study
    J Neurol NeuroSurg Psychiatry 2005 Feb;76(2):206-11

  6. Recent NeuroPathological findings in MS - implications for diagnosis and therapy
    J Neurol 2004 Sep;251 Suppl 4:IV2-IV5

  7. Neuronal cell injury precedes Brain Atrophy in Multiple Sclerosis
    Neurology 2004 Feb 24;62(4):624-7

  8. Thalamic involvement in Multiple Sclerosis: a Diffusion-weighted Magnetic Resonance Imaging study
    J NeuroImaging 2003 Oct;13(4):307-14

  9. Axonal degeneration and progressive Neurologic disability in Multiple Sclerosis
    NeuroTox Res 2003;5(1-2):157-64

  10. Multiple Sclerosis: NeuroFilament light chain AntiBodies are correlated to Cerebral Atrophy
    Neurology 2003 Jan 28;60(2):219-23

  11. Measurement of Atrophy in Multiple Sclerosis: pathological basis, methodological aspects and clinical relevance
    Brain 2002 Aug;125(Pt 8):1676-1695

  12. Gray matter T2 HypoIntensity is related to plaques and Atrophy in Multiple Sclerosis Brains
    J Neurol Sci 2001 Mar 15;185(1):19-26

  13. Transected Neurites, Apoptotic Neurons, and Reduced Inflammation in Cortical Multiple Sclerosis lesions
    Ann Neurol 2001 Sep;50(3):389-400

  14. Electrically active Axons degenerate when exposed to Nitric Oxide
    Ann Neurol 2001 Apr;49(4):470-6

  15. MTR Histogram analysis of Gray Matter in Relapsing/Remitting Multiple Sclerosis
    AJNR Am J NeuroRadiol 2001 Mar;22(3):470-475

  16. Relating Axonal injury to functional recovery in MS
    Neurology 2000 Jan 11;54(1):236-9

  17. Bulk List Must Read
    Anoxic and Ischemic injury of Myelinated CNS Axons

  18. Axonal loss in Multiple Sclerosis lesions: MRI insights into substrates of disability
    Ann Neurol 1999 Nov;46(5):747-54

  1. Correlating Magnetic Resonance Imaging markers of Axonal injury and DeMyelination in motor impairment secondary to Stroke and Multiple Sclerosis
    Magn Reson Imaging 2000 May;18(4):369-78

  2. Proton Magnetic Resonance Spectroscopy in Primary and Secondary/Progressive Multiple Sclerosis
    NMR Biomed 2000 Apr;13(2):57-63

  3. Axonal changes in chronic DeMyelinated Cervical Spinal Cord plaques
    Brain 2000 Feb;123 ( Pt 2):308-17

  4. In vivo evidence for Axonal dysfunction remote from focal Cerebral DeMyelination of the type seen in Multiple Sclerosis
    Brain 1999 Oct;122 ( Pt 10):1933-9

  5. Neuronal damage in T1-HypoIntense Multiple Sclerosis lesions demonstrated in vivo using Proton Magnetic Resonance Spectroscopy
    Ann Neurol 1999 Jul;46(1):79-87

  6. Axonal damage in Multiple Sclerosis plaques: a combined Magnetic Resonance Imaging and 1H-Magnetic Resonance Spectroscopy study
    J Neurol Sci 2001 Jan 1;182(2):143-150

  7. Axonal damage induced by CerebroSpinal Fluid from patients with Relapsing/Remitting Multiple Sclerosis
    J NeuroImmunol 2000 Apr 3;104(1):58-67

  8. Evidence of Axonal Damage in the Early Stages of Multiple Sclerosis and Its Relevance to Disability
    Arch Neurol 2001 Jan;58(1):65-70

  9. PlasMinogen Activators in Multiple Sclerosis lesions: Implications for the inflammatory response and Axonal damage
    Brain 2001 Oct;124(Pt 10):1978-88

  10. Acute Axonal damage in Multiple Sclerosis is most extensive in early disease stages and decreases over time
    Brain 2002 Oct;125(Pt 10):2202-12

  11. Multiple Sclerosis and chronic AutoImmune Encephalomyelitis: a comparative quantitative study of Axonal injury in active, inactive, and ReMyelinated lesions
    Am J Pathol 2000 Jul;157(1):267-76